Apoptosis Genes

Apoptosis Genes by James W. Wilson, published by Springer US on December 16, 2012, is a comprehensive exploration of the mechanisms behind programmed cell death, a vital process for maintaining cellular health and balance. This edition, spanning 310 pages, delves into the intricate interactions between genes and proteins that govern apoptosis, highlighting recent advancements in the field that may pave the way for innovative therapeutic approaches.
Readers will find a detailed overview of key genes involved in apoptosis regulation, along with insights into future research directions and potential clinical applications. The book addresses the significance of apoptosis in various medical contexts, including cancer, autoimmune diseases, and neurodegenerative disorders. Apoptosis Genes serves as a valuable resource for researchers and professionals in the fields of biochemistry, genetics, and oncology, providing a focused examination of how understanding apoptosis can influence treatment strategies and disease management.
Official synopsis Publisher
Apoptosis, or programmed cell death, is a natural process by which damaged or unwanted cells are dismantled in an orderly and atraumatic fashion. It is of critical importance in development, homeostasis, and cell population control. Research over the last decade is now enabling scientists to comprehend how genes and the protein products interact to control apoptosis. This has led to the current position where researchers may be able to directly modify the action of key proteins through gene therapy and antisense oligonucleotides.
Apoptosis Genes presents a current overview of key genes involved in the control of apoptosis research together with thoughts on future prospects and clinical applications. While there are several books written on apoptosis, Apoptosis Genes deals specifically with the regulation of apoptosis. Given the increased interest in the role of apoptosis genes in disease processes, this work will be useful to researchers investigating cancer, autoimmune disease, viral infection, cardiovascular disease, neurodegenerative disorders, AIDS, osteoporosis, and aging.
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